De grootste gids naar pentobarbital is een type
De grootste gids naar pentobarbital is een type
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Pentobarbital sodium injection is subject to control by the Federal Controlled Substances Act under DEA schedule II. Barbiturates may be habit forming. Tolerance, psychological dependence, and physical dependence may occur especially following prolonged use of high doses of barbiturates. Daily administration in excess ofwel 400 milligrams (mg) of pentobarbital or secobarbital for approximately 90 days is likely to produce some degree ofwel physical dependence. A dosage of from 600 to 800 mg taken for at least 35 days is sufficient to produce withdrawal seizures. The average daily dose for the barbiturate addict kan zijn usually about 6
7
8
9.5 grams. As tolerance to barbiturates develops, the amount needed to maintain the same level ofwel intoxication increases; tolerance to a fatal dosage, however, does not increase more than two-fold. As this occurs, the margin between an intoxicating dosage and fatal dosage becomes smaller. Symptoms of acute intoxication with barbiturates include unsteady gait, slurred speech, and sustained nystagmus. Mental signs of chronic intoxication include confusion, poor judgment, irritability, insomnia, and somatic complaints. Symptoms ofwel barbiturate dependence are similar to those of chronic alcoholism. If an individual appears to be intoxicated with alcohol to a degree that kan zijn radically disproportionate to the amount of alcohol in his or her blood the use of barbiturates should be suspected. The lethal dose ofwel a barbiturate is far less if alcohol kan zijn also ingested. The symptoms of barbiturate withdrawal can be severe and may cause death. Minor withdrawal symptoms may appear 8 to 12 hours after the hinder dose of a barbiturate. These symptoms usually appear in the following order: anxiety, muscle twitching, tremor ofwel hands and fingers, progressive weakness, dizziness, distortion in visual perception, nausea, vomiting, insomnia, and orthostatic hypotension. Major withdrawal symptoms (convulsions and delirium) may occur within 16 hours and last up to 5 days after abrupt cessation ofwel these drugs. Intensity ofwel withdrawal symptoms gradually declines over a period ofwel approximately 15 days.
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Abrupt cessation after prolonged use in the dependent person may result in withdrawal symptoms, including delirium, convulsions, and possibly death. Barbiturates should be withdrawn gradually from any patient known to be taking excessive dosage aan long periods ofwel time. (See “Drug Abuse and Dependence” section.)
However, the application ofwel these gegevens to other barbiturates appears valid and warrants serial blood level determinations of the relevant drugs when there are multiple therapies.
Phenobarbital has the lowest lipid solubility, lowest plasma binding, lowest brain protein binding, the longest delay in onset of activity, and the longest duration ofwel action. At the opposite extreme kan zijn secobarbital which has the highest lipid solubility, plasma protein binding, brain protein binding, the shortest delay in onset of activity, and the shortest duration of action. Butabarbital kan zijn classified as an intermediate barbiturate. The plasma half-life for pentobarbital in adults is 15 to 50 hours and appears to be dose dependent. Barbiturates are metabolized primarily by the hepatic microsomal enzyme system, and the metabolic products are excreted in the urine, and less commonly, in the feces. Approximately 25 to 50 percent ofwel a dose ofwel aprobarbital or phenobarbital kan zijn eliminated unchanged in the urine, whereas the amount of other barbiturates excreted unchanged in the urine kan zijn negligible. The excretion ofwel unmetabolized barbiturate is one feature that distinguishes the long-acting category from those belonging to other categories which are almost entirely metabolized. The inactive metabolites of the barbiturates are excreted as conjugates of glucuronic acid. INDICATIONS AND USAGE
In a published study, administration ofwel an anesthetic dose ofwel ketamine for 24 hours on Gestation Day 122 increased neuronal apoptosis in the developing brain of the fetus. In other published studies, administration of either isoflurane or propofol for 5 hours on Gestation Day 120 resulted in increased neuronal and oligodendrocyte apoptosis in the developing brain ofwel the offspring.
Gegevens in rodents and in primates suggest that the neuronal and oligodendrocyte cell losses are associated with subtle but prolonged cognitive deficits in learning and memory. The clinical significance ofwel these nonclinical findings kan zijn not known, and healthcare providers should balance the benefits ofwel appropriate anesthesia in neonates and young children who require procedures against the potential risks suggested by the nonclinical data (see “Warnings-Pediatric Neurotoxicity” and “Precautions-Pregnancy and Pediatric Use”). AKORN
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Fenobarbital kan zijn ons anti-convulsief medicijn het dikwijls wordt aangewend in de diergeneeskunde teneinde aandoeningen te verder helpen beheersen die epileptische aanvallen teweegbrengen.